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Latest
Advancement in Anti-Aging Skin Care- Nur76 Anti-Aging
breakthrough!
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News
report:
St.
Paul, MN. June 1, 2004 - For hundreds of years man has
been in search of the Fountain of Youth; a magical potion
that can maintain one’s youth indefinitely. Yet,
still today facial wrinkles are the markings of a person
in the later years of their life.
More
and more people are turning to anti-wrinkle creams to
substitute for the Fountain of Youth. Many anti-wrinkle
creams claim to smoothen out the skin. What people do
not realize is that most of these creams use AHAs (alpha
hydroxy acids), also known as fruit acids. These chemicals
work by peeling off the outer layer of the skin to reveal
the skin beneath.
However,
most dermatologists are concerned that AHAs could in fact
cause long-term damage instead of rejuvenating the skin.
The European Commission is so concerned about these AHA’s
that it is considering placing warning labels on all creams
containing these chemicals.
A
clinically proven compound to remove wrinkles without
harmful side effects is Acetyl Hexapeptide (Argireline).
This peptide is a powerful non toxic ingredient that can
dramatically reduce fine lines and wrinkles around the
eyes, mouth, and forehead.
Argireline
reduces the amount of nerve stimulation to the facial
muscles, thus causing the area to smoothen and lessen
the appearance of wrinkles. Nur76 Serum (the latest serum
in anti-aging development) is one of the few anti-aging
products that contains the Argireline.
This
compound not only prevents the effects of aging of skin,
but also has been clinically proven to remove existing
lines and wrinkles, particularly in the forehead and eye
area. ”Argireline not only reverses the aging process,
but is also known to prevent further aging of the skin.
In
today’s youth oriented culture a younger appearance
has greater status. Cosmetic surgery and injections are
glorified in the media everyday, without mentioning the
cost and health risks. Nur76 Serum can help give you that
extra youthful glow, that smoother skin, without costly
and dangerous surgery or paralyzing injections of what
is essentially botulism toxin.”
“The
inventors presented several scientific studies to the
Life Extension staff and showed stunning before-and-after
photographs demonstrating this liquid’s efficacy.
Finally, it seemed, there was a topical liquid that produced
many of the effects of injections without the pain and
huge expense.”
“For
the first time, a patent-pending delivery system has been
developed that enables very large molecules (such as collagen)
to pass through the upper layer of skin. When this new
trans dermal system is combined with collagen, the collagen
molecules are delivered directly to the third layer of
the epidermis—an effect achieved previously only
by injection.”
Latest
news on scientific breakthrough newsweek 2005
By
Mary Carmichael and Jennifer Barrett Ozols
Newsweek
Jan.
17 issue - Though death is still as inevitable as taxes,
future generations may age more slowly and live significantly
longer. Here are five scientists in the vanguard of research,
offering new insights into the biochemistry of aging—and
opening the door for life-lengthening drugs. Their approaches
vary, but they share the belief that the human life span
is not fixed.
Enhanced:TARGETED
GENES ARE MORE ACTIVE IN FIGHTING AGING
The
"guess your age" booth at a carnival isn't often
exactly right. But it's not usually as off-base as Cynthia
Kenyon's colleagues. A few years ago Kenyon, a molecular
geneticist, had one of her grad students cart a tray of
worms around her lab, asking people how old they thought
the worms were. Most said about 5 days. What they didn't
know was that Kenyon had tinkered with the worms' genes.
The squirmy creatures had the perfect health of 5-day-olds,
but they were 144 days old—six times their normal
life span.
Over
the last decade, Kenyon's continuing work has shown that
"you can make huge changes in life span so easily"—in
worms, at least—by changing hormone levels and enhancing
the effects of fewer than 100 genes. Some of the target
genes produce antioxidants; some make natural microbicides;
some are involved in transporting fats throughout the
body, and some, called chaperones, "keep the cell
components in good working order," says Kenyon. What
they all have in common is their effect on aging. The
more active the genes, in general, the longer an organism
is likely to live.
When
Kenyon's work with worm genes was first published in 1993,
skeptics predicted it wouldn't translate well to humans.
One hundred forty-four days might be ancient for a worm,
but a far more complex human being can already expect
to live about 200 times longer than that. Scientists still
don't know exactly why the life spans are so different,
much less what a change in a worm's life span might mean
for a person's. Nonetheless, much of the cellular machinery
in worms closely resembles that in higher mammals. That
finding has opened the door for a neutraceutical company,
which is trying to develop a drug that would yield the
same kind of results as Kenyon's genetic tampering. "I'm
not saying that with a few changes humans could be immortal,"
she says. "But it'd be like looking at an 80-year-old
and thinking he was 40." Who could object to that?
Stressed:CHRONIC
TENSION MAKES CELLS DETERIORATE FASTER
If
you've ever blamed stress for new wrinkles or gray hairs,
you may have been right. "As a society, we have a
deeply held belief that life stress causes premature aging,
but there's actually been very little empirical evidence
to show this," says Elisa Epel, assist-ant professor
of psychiatry at the University of California, San Francisco.
Until
now. In a UCSF-led study published this past fall in Proceedings
of the National Academy of Sciences, Epel and her colleagues
found that chronic stress—or even the perception
of stress—significantly shortened the length of
telomeres, the tips of chromosomes within cells that can
be used as a measure of the cells' aging process. The
shorter the telomere, the shorter the cell's life span
and the faster the body's deterioration. As more cells
die, the effects of aging kick in: muscles weaken, skin
wrinkles and eyesight and hearing worsen.
Epel
and her colleagues studied 39 women between the ages of
20 and 50 with children suffering from serious chronic
conditions, like cerebral palsy, and compared them with
19 mothers in the same age group with healthy children.
The longer a woman had been caring for a sick child, the
shorter her telomeres—and the greater her oxidative
stress (a process that releases DNA-damaging free radicals).
But
what startled researchers more was that the most profound
differences were tied to the women's perceptions of how
much emotional strain they were under, regardless of whether
their children were healthy or sick. When compared with
the women with the lowest perceived stress levels, women
in both groups who described themselves as having the
highest stress levels had telomeres equivalent to someone
10 years older.
While
Epel acknowledges that more studies need to be done to
confirm her findings, she says the results could have
positive implications. "Now that we think we can
see intracellular damage from stress, people might weigh
the importance of positive mental health more heavily,"
she says, adding that there is "absolutely"
hope that the DNA damage is reversible. "Lifestyle
changes—and learning to cope well with stress—could
potentially improve your quality of life, your mood and
your longevity.
"Restricted:A
TOUGH LIMIT ON CALORIE INTAKE MAY SLOW AGING
Leonard Guarente didn't come up with the trick of calorie
restriction, or strictly limiting nutrients to achieve
longer life. And the idea sounded crazy back in 1986,
when Guarente first proposed to study the biology of aging
via calorie restriction. Aging was seen as too complex
a topic for molecular biologists, and the effect of calorie
restriction on aging, though detailed in scientific literature
since the 1930s, was even more poorly understood. Guarente's
colleagues called him "bonkers," but he didn't
care: "I wanted to work on something risky,"
he says. "Besides, I had just gotten tenure, and
at that point they couldn't get rid of me. "They
certainly wouldn't want to now. Guarente is not the least
bit bonkers—and, unbeknown to his colleagues at
the time, he wasn't even the only scientist thinking about
the molecular biology of calorie restriction. In the last
decade, researchers have made great strides in understanding
why a sudden drop in calorie intake can kick up the activity
of a gene called SIR2 and prolong life in simple organisms.
At
the head of the class are Guarente and a Harvard researcher
named David Sinclair, both of whom are focusing on sirtuins,
the family of proteins produced by SIR2 or its mammalian
analogue, SIRT1. Guarente's lab has unraveled many of
the basic molecular processes behind SIR2. For instance,
a natural chemical called NADH can inhibit sirtuins' effects;
Guarente's lab has determined that yeast with lower NADH
levels lives longer. Sinclair's work has a slightly different
focus—resveratrol, the chemical he has connected
to calorie restriction's effects. (It's better known as
the major reason red wine is touted as healthful.) Sinclair's
work at Harvard has shown that heavy doses of resveratrol
can prolong life span in yeast by 70 percent. Still another
scientist, Marc Tatar, has garnered similar results in
fruit flies.
The
fact that calorie restriction works isn't all that surprising
from an evolutionary point of view. In fact, calorie restriction
is an extremely effective strategy for survival during
lean times, when it's an imperative, not a choice. "Let's
imagine I had a gene that could allow me to suspend reproduction
and slow down aging during a famine," says Guarente.
"When the famine ends, I'll still be around to reproduce."
As a result, he adds, "every animal we know can do
this."
Including
humans, of course. But since few people particularly want
to limit their calories drastically (least of all Americans),
Guarente is searching for a pill that will have the same
effect the same company building on Kenyon's work, is
also using Guarente's—which means, someday, humans
may reap the benefits of calorie restriction without even
having to say the word diet. Sinclair has a competing
company called Sirtris. He expects to get his drugs into
clinics in just five years. Until then, he'll be drinking
one glass of red wine a day—and toasting to what
he hopes will be a huge success.
Supplemented:TWO
CHEMICALS MADE OLD RATS NEW
Five
years ago Bruce Ames called his son, a computer executive
in New York, with some exciting news. "I told him,
'We're changing old rats to new rats!' " recalls
Ames, a senior scientist at Children's Hospital Oakland
Research Institute in California. His son was not impressed.
"Let me know when you change old people to young
rats," he said. Such human-to-animal transformations
are still confined to the minds of sarcastic sons and
science-fiction writers, but researchers are getting closer
to replicating Ames's rat results in humans.
In
studies published in Proceedings of the National Academy
of Sciences in 2002, Ames and his colleagues fed older
rats two chemicals normally found in the body's cells
(and also sold as nutritional supplements): acetyl-L-carnitine
and alphalipoic acid. Not only did the rats perform better
on problem-solving and memory tests, but they moved around
with more ease and energy.
Researchers
determined that the combination of chemicals had improved
the function of mitochondria, organelles that serve as
a cell's main energy source. Ames formed a company called
Juvenon to license the combination of cell-rejuvenating
supplements (also sold separately at several health stores).
The company plans to begin human trials soon to evaluate
the cognitive effects of the dual supplements. In the
meantime, Ames, who chairs Juvenon's scientific advisory
board but gets no proceeds from the company, is overseeing
lab research on human cells in tissue culture. In one
study, Berkeley researchers found that lipoic acid protected
the cell from oxidation when iron or hydrogen peroxide
was added.
Now
he hopes to replicate those results in human subjects.
Other studies have already linked unhealthy mitochondria
to Alzheimer's, Parkinson's, type 2 diabetes and other
degenerative diseases, so reversing or repairing decay
in mitochondria could help to stave off the age-related
diseases. "I'm hoping we can add a few years to people's
lives," says Ames, who's 76. "I think we can."
NewsweekJan.
17 issue - VIENNA, VA.: I've been very overweight for
eight years; before that, I was a normal size. Is it possible
at 60 to get back to my old self—or is that a dream?
DR. HOWARD LEWINE: No, it is not a dream to get fit and
trim at 60. But it probably won't be easy to get there.
Assuming that you are otherwise quite healthy, I recommend
exercise as your first priority. If you have been very
sedentary, then you want to check with your doctor before
starting a physically vigorous program. Once you are medically
clear, start with about 10 minutes to 15 minutes of slow-paced
exercise, like walking or water aerobics, once or twice
daily. Work up to a faster heart rate. As exercise becomes
a regular part of your daily routine, start cutting calories.
This is not a diet—it is eating less each day until
your caloric intake becomes less than your daily energy
expenditure. Weight loss will follow.
LOUISVILLE,
KY.: Is there any way to avoid having your voice age?
Even on the telephone, the voice is a dead giveaway that
a person is old. Why?
As we age, the cartilage support for the vocal cords becomes
stiffer, decreasing mobility of the cords. The vocal cords
and the muscles of the larynx also lose tone, causing
a gap between the vocal cords when they try to close completely.
The age at which this can occur varies. Although you can't
slow down these changes, you can help your voice stay
strong by exercising regularly and avoiding smoking. If
you are persistently hoarse, you might also consider whether
stomach-acid reflux is irritating the vocal cords.
SANDY, UTAH: What is your opinion on the severe calorie-restriction
diet being promoted by some people to slow the aging process?
We don't have the evidence yet that significant calorie
restriction in humans will extend life or slow aging,
but the studies in animals are quite compelling. Mice
that are allowed to eat as much as they want die much
sooner than mice fed a diet with 35 percent fewer calories
daily. Preliminary studies in primates also suggest healthier
aging by eating a calorie-restricted diet. I personally
believe that future research will prove calorie-restriction
benefits independent of weight loss for humans. But those
restricting calories need to make sure that they are getting
enough protein and micro nutrients.